Open Access. Powered by Scholars. Published by Universities.®
- Discipline
- Keyword
-
- Animals (12)
- Mice (11)
- Humans (6)
- Genetic (5)
- Knockout (5)
-
- Mice, Knockout (5)
- Base Sequence (4)
- DNA Nucleotidylexotransferase (4)
- Gene Targeting (4)
- Inbred C57BL (4)
- Mice, Inbred C57BL (4)
- Molecular Sequence Data (4)
- T-Lymphocytes (4)
- Cultured (3)
- Epithelial Sodium Channels (3)
- Gene Expression Regulation (3)
- Globins (3)
- Heterozygote (3)
- Sodium Channels (3)
- Adaptor Proteins (2)
- Adaptor Proteins, Signal Transducing (2)
- Animal (2)
- Cell Line (2)
- Cells (2)
- Cells, Cultured (2)
- Chimera (2)
- DNA Primers (2)
- Disease Models (2)
- Disease Models, Animal (2)
- Enzymologic (2)
Articles 1 - 30 of 80
Full-Text Articles in Obstetrics and Gynecology
Gene Modification Via "Plug And Socket" Gene Targeting., J. Lewis, Baoli Yang, P. Detloff, O. Smithies
Gene Modification Via "Plug And Socket" Gene Targeting., J. Lewis, Baoli Yang, P. Detloff, O. Smithies
Baoli Yang
No abstract provided.
A Common Human Beta Globin Splicing Mutation Modeled In Mice., J. Lewis, Baoli Yang, R. Kim, H. Sierakowska, R. Kole, O. Smithies, N. Maeda
A Common Human Beta Globin Splicing Mutation Modeled In Mice., J. Lewis, Baoli Yang, R. Kim, H. Sierakowska, R. Kole, O. Smithies, N. Maeda
Baoli Yang
The betaIVS-2-654 C-->T mutation accounts for approximately 20% of beta thalassemia mutations in southern China; it causes aberrant RNA splicing and leads to beta0 thalassemia. To provide an animal model for testing therapies for correcting splicing defects, we have used the "plug and socket" method of gene targeting in murine embryonic stem cells to replace the two (cis) murine adult beta globin genes with a single copy of the human betaIVS-2-654 gene. No homozygous mice survive postnatally. Heterozygous mice carrying this mutant gene produce reduced amounts of the mouse beta globin chains and no human beta globin, and have …
T-Cell Specific Avian Tdt: Characterization Of The Cdna And Recombinant Enzyme., Baoli Yang, K. Gathy, M. Coleman
T-Cell Specific Avian Tdt: Characterization Of The Cdna And Recombinant Enzyme., Baoli Yang, K. Gathy, M. Coleman
Baoli Yang
A cDNA clone coding for avian terminal deoxynucleotidyl transferase (TdT) has been isolated and sequenced. The size of this cDNA was 2545 bp with an open reading frame of 1521 bp and a predicted translation product of 58 kDa. Comparison of this TdT sequence with other known TdT sequences has revealed a very high degree of homology at both the DNA and predicted amino acid levels. The chicken TdT cDNA was expressed in a bacterial system and the protein was purified by affinity chromatography. The purified recombinant enzyme, with a specific activity of approximately 1700 U/mg protein, was significantly less …
Fetal Hemorrhage And Platelet Dysfunction In Slp-76-Deficient Mice., J. Clements, J. Lee, B. Gross, Baoli Yang, J. Olson, A. Sandra, S. Watson, S. Lentz, G. Koretzky
Fetal Hemorrhage And Platelet Dysfunction In Slp-76-Deficient Mice., J. Clements, J. Lee, B. Gross, Baoli Yang, J. Olson, A. Sandra, S. Watson, S. Lentz, G. Koretzky
Baoli Yang
The adapter protein SLP-76 is expressed in T lymphocytes and hematopoietic cells of the myeloid lineage, and is known to be a substrate of the protein tyrosine kinases that are activated after ligation of the T-cell antigen receptor. Transient overexpression of SLP-76 in a T-cell line potentiates transcriptional activation after T-cell receptor ligation, while loss of SLP-76 expression abrogates several T-cell receptor-dependent signaling pathways. Mutant mice that lack SLP-76 manifest a severe block at an early stage of thymocyte development, implicating SLP-76 in signaling events that promote thymocyte maturation. While it is clear that SLP-76 plays a key role in …
Syntaxin 4 Heterozygous Knockout Mice Develop Muscle Insulin Resistance., C. Yang, K. Coker, J. Kim, S. Mora, D. Thurmond, A. Davis, Baoli Yang, R. Williamson, G. Shulman, J. Pessin
Syntaxin 4 Heterozygous Knockout Mice Develop Muscle Insulin Resistance., C. Yang, K. Coker, J. Kim, S. Mora, D. Thurmond, A. Davis, Baoli Yang, R. Williamson, G. Shulman, J. Pessin
Baoli Yang
To investigate the physiological function of syntaxin 4 in the regulation of GLUT4 vesicle trafficking, we used homologous recombination to generate syntaxin 4-knockout mice. Homozygotic disruption of the syntaxin 4 gene results in early embryonic lethality, whereas heterozygous knockout mice, Syn4(+/-), had normal viability with no significant impairment in growth, development, or reproduction. However, the Syn4(+/-) mice manifested impaired glucose tolerance with a 50% reduction in whole-body glucose uptake. This defect was attributed to a 50% reduction in skeletal muscle glucose transport determined by 2-deoxyglucose uptake during hyperinsulinemic-euglycemic clamp procedures. In parallel, insulin-stimulated GLUT4 translocation in skeletal muscle was also …
The Ubiquitin Ligase Adaptor Ndfip1 Regulates T Cell-Mediated Gastrointestinal Inflammation And Inflammatory Bowel Disease Susceptibility., H. Ramon, C. Riling, J. Bradfield, Baoli Yang, H. Hakonarson, P. Oliver
The Ubiquitin Ligase Adaptor Ndfip1 Regulates T Cell-Mediated Gastrointestinal Inflammation And Inflammatory Bowel Disease Susceptibility., H. Ramon, C. Riling, J. Bradfield, Baoli Yang, H. Hakonarson, P. Oliver
Baoli Yang
Nedd4 family interacting protein 1 (Ndfip1) is an adaptor protein that regulates Itch, an E3 ubiquitin ligase that ubiquitylates JunB, thereby preventing interleukin (IL)-4 and IL-5 production. Mice lacking Ndfip1 or Itch develop T helper type 2 (T(H)2)-mediated inflammation in the skin and lungs and die prematurely. In this study we show that Ndfip1-/- mice also develop inflammation in the gastrointestinal tract. Inflammation is characterized by infiltration of eosinophils and T cells and is accompanied by a failure to gain weight. T cells are both necessary and sufficient for eosinophil recruitment and inflammation. This is because Ndfip1-/- T cells become …
Disruption Of The Beta Subunit Of The Epithelial Na+ Channel In Mice: Hyperkalemia And Neonatal Death Associated With A Pseudohypoaldosteronism Phenotype., F. Mcdonald, Baoli Yang, R. Hrstka, H. Drummond, D. Tarr, P. Mccray, J. Stokes, M. Welsh, R. Williamson
Disruption Of The Beta Subunit Of The Epithelial Na+ Channel In Mice: Hyperkalemia And Neonatal Death Associated With A Pseudohypoaldosteronism Phenotype., F. Mcdonald, Baoli Yang, R. Hrstka, H. Drummond, D. Tarr, P. Mccray, J. Stokes, M. Welsh, R. Williamson
Baoli Yang
The epithelial Na+ channel (ENaC) is composed of three homologous subunits: alpha, beta and gamma. We used gene targeting to disrupt the beta subunit gene of ENaC in mice. The betaENaC-deficient mice showed normal prenatal development but died within 2 days after birth, most likely of hyperkalemia. In the -/- mice, we found an increased urine Na+ concentration despite hyponatremia and a decreased urine K+ concentration despite hyperkalemia. Moreover, serum aldosterone levels were increased. In contrast to alphaENaC-deficient mice, which die because of defective lung liquid clearance, neonatal betaENaC deficient mice did not die of respiratory failure and showed only …
A Mouse Model For Beta 0-Thalassemia., Baoli Yang, S. Kirby, J. Lewis, P. Detloff, N. Maeda, O. Smithies
A Mouse Model For Beta 0-Thalassemia., Baoli Yang, S. Kirby, J. Lewis, P. Detloff, N. Maeda, O. Smithies
Baoli Yang
We have used a "plug and socket" targeting technique to generate a mouse model of beta 0-thalassemia in which both the b1 and b2 adult globin genes have been deleted. Mice homozygous for this deletion (Hbbth-3/Hbbth-3) die perinatally, similar to the most severe form of Cooley anemia in humans. Mice heterozygous for the deletion appear normal, but their hematologic indices show characteristics typical of severe thalassemia, including dramatically decreased hematocrit, hemoglobin, red blood cell counts, mean corpuscular volume, mean corpuscular hemoglobin, and mean corpuscular hemoglobin concentration, as well as dramatically increased reticulocyte counts, serum bilirubin concentrations, and red cell distribution …
Appropriate Tissue- And Cell-Specific Expression Of A Single Copy Human Angiotensinogen Transgene Specifically Targeted Upstream Of The Hprt Locus By Homologous Recombination., B. Cvetkovic, Baoli Yang, R. Williamson, C. Sigmund
Appropriate Tissue- And Cell-Specific Expression Of A Single Copy Human Angiotensinogen Transgene Specifically Targeted Upstream Of The Hprt Locus By Homologous Recombination., B. Cvetkovic, Baoli Yang, R. Williamson, C. Sigmund
Baoli Yang
Development of experimental models by genetic manipulation in mice has proven to be very useful in determining the significance of particular genes in the development of or susceptibility to hypertension. Advances in molecular genetics, transgenic mouse technology, and physiological measurements in mice provided an opportunity to go a step further and develop models to analyze the physiological significance of specific gene variants potentially causing hypertension. In this report, we describe the development of a human angiotensinogen transgenic mouse model generated by targeting the human angiotensinogen gene upstream of the mouse HPRT locus by homologous recombination. The main benefit of this …
Requirement For The Leukocyte-Specific Adapter Protein Slp-76 For Normal T Cell Development., J. Clements, Baoli Yang, S. Ross-Barta, S. Eliason, R. Hrstka, R. Williamson, G. Koretzky
Requirement For The Leukocyte-Specific Adapter Protein Slp-76 For Normal T Cell Development., J. Clements, Baoli Yang, S. Ross-Barta, S. Eliason, R. Hrstka, R. Williamson, G. Koretzky
Baoli Yang
The leukocyte-specific adapter molecule SLP-76 (Src homology 2 domain-containing leukocyte protein of 76 kilodaltons) is rapidly phosphorylated on tyrosine residues after receptor ligation in several hematopoietically derived cell types. Mice made deficient for SLP-76 expression contained no peripheral T cells as a result of an early block in thymopoiesis. Macrophage and natural killer cell compartments were intact in SLP-76-deficient mice, despite SLP-76 expression in these lineages in wild-type mice. Thus, the SLP-76 adapter protein is required for normal thymocyte development and plays a crucial role in translating signals mediated by pre-T cell receptors into distal biochemical events.
The Effect Of Monoclonal Anti-Hcg Antibodies On The Bioactivity And The Binding With Receptor Of Hcg And Lh, Baoli Yang, F. Zhou, Y. Yang
The Effect Of Monoclonal Anti-Hcg Antibodies On The Bioactivity And The Binding With Receptor Of Hcg And Lh, Baoli Yang, F. Zhou, Y. Yang
Baoli Yang
No abstract provided.
The Mammalian Sodium Channel Bnc1 Is Required For Normal Touch Sensation., M. Price, G. Lewin, S. Mcilwrath, C. Cheng, J. Xie, P. Heppenstall, C. Stucky, A. Mannsfeldt, T. Brennan, H. Drummond, J Qiao, C. Benson, D Tarr, R Hrstka, Baoli Yang, R. Williamson, M. Welsh
The Mammalian Sodium Channel Bnc1 Is Required For Normal Touch Sensation., M. Price, G. Lewin, S. Mcilwrath, C. Cheng, J. Xie, P. Heppenstall, C. Stucky, A. Mannsfeldt, T. Brennan, H. Drummond, J Qiao, C. Benson, D Tarr, R Hrstka, Baoli Yang, R. Williamson, M. Welsh
Baoli Yang
Of the vertebrate senses, touch is the least understood at the molecular level The ion channels that form the core of the mechanosensory complex and confer touch sensitivity remain unknown. However, the similarity of the brain sodium channel 1 (BNC1) to nematode proteins involved in mechanotransduction indicated that it might be a part of such a mechanosensor. Here we show that disrupting the mouse BNC1 gene markedly reduces the sensitivity of a specific component of mechanosensation: low-threshold rapidly adapting mechanoreceptors. In rodent hairy skin these mechanoreceptors are excited by hair movement. Consistent with this function, we found BNC1 in the …
Nedd4 And Nedd4-2: Closely Related Ubiquitin-Protein Ligases With Distinct Physiological Functions., Baoli Yang, S. Kumar
Nedd4 And Nedd4-2: Closely Related Ubiquitin-Protein Ligases With Distinct Physiological Functions., Baoli Yang, S. Kumar
Baoli Yang
The Nedd4 (neural precursor cell-expressed developmentally downregulated gene 4) family of ubiquitin ligases (E3s) is characterized by a distinct modular domain architecture, with each member consisting of a C2 domain, 2-4 WW domains, and a HECT-type ligase domain. Of the nine mammalian members of this family, Nedd4 and its close relative, Nedd4-2, represent the ancestral ligases with strong similarity to the yeast, Rsp5. In Saccharomyces cerevisiae Rsp5 has a key role in regulating the trafficking, sorting, and degradation of a large number of proteins in multiple cellular compartments. However, in mammals the Nedd4 family members, including Nedd4 and Nedd4-2, appear …
Identification Of A Tripartite Basal Promoter Which Regulates Human Terminal Deoxynucleotidyl Transferase Gene Expression., D. Bhaumik, Baoli Yang, T. Trangas, J. Bartlett, M. Coleman, D. Sorscher
Identification Of A Tripartite Basal Promoter Which Regulates Human Terminal Deoxynucleotidyl Transferase Gene Expression., D. Bhaumik, Baoli Yang, T. Trangas, J. Bartlett, M. Coleman, D. Sorscher
Baoli Yang
In order to locate the promoter region of the human terminal deoxynucleotidyl transferase gene, serially truncated segments of the 5'-flanking region of the gene were cloned into a chloramphenicol acetyltransferase reporter vector. Transient transfection analyses of the terminal transferase-reporter gene constructs identified the basal promoter region within -34 to +40 base pairs relative to the transcription start site. Three promoter elements were defined in this region. The primary element is within 34 base pairs upstream of the transcription start site. The CAP site is 62 base pairs upstream of the translation start site. The secondary element involves sequences around the …
Regulation Of Terminal Deoxynucleotidyl Transferase Gene Expression In Mice And Men., M. Coleman, Baoli Yang, D. Sorscher
Regulation Of Terminal Deoxynucleotidyl Transferase Gene Expression In Mice And Men., M. Coleman, Baoli Yang, D. Sorscher
Baoli Yang
A nontemplate directed DNA polymerase, terminal deoxynucleotidyl transferase (terminal transferase) is expressed in a tissue-specific and development stage-specific manner. Its enzymatic properties and tissue localization have implicated the protein in development of normal immune function. Significant progress has been made in understanding the enzymology and important domains of this protein. More recently, studies have focused on regulation of the gene that codes for the protein in mice and humans. The murine gene has yielded to these studies more readily than the human gene. A murine basal promoter element has been identified along with several trans-acting protein factors that may regulate …
Mutational Analysis Of Residues In The Nucleotide Binding Domain Of Human Terminal Deoxynucleotidyl Transferase., Baoli Yang, K. Gathy, M. Coleman
Mutational Analysis Of Residues In The Nucleotide Binding Domain Of Human Terminal Deoxynucleotidyl Transferase., Baoli Yang, K. Gathy, M. Coleman
Baoli Yang
Human terminal deoxynucleotidyl transferase (TdT) was overexpressed in a baculovirus system. The pure recombinant enzyme was identical in size, activity, kinetic constants, and metal effects to native enzyme. Three amino acids, within either the putative nucleotide binding domain and part of a DNA polymerase consensus sequence, YGDTDSLF, or a TdT consensus sequence, GGFRRGK, were altered by site-directed mutagenesis. The four mutant forms of terminal transferase were also overexpressed in the baculovirus expression system and purified from Trichoplusia ni larvae by a monoclonal antibody affinity column and compared with wild-type enzyme with respect to thermostabilities, secondary structure, metal effects, and kinetic …
Diminished Cytokine And Chemokine Expression In The Central Nervous System Of Gmf-Deficient Mice With Experimental Autoimmune Encephalomyelitis, Asgar Zaheer, Shailendra Sahu, Yanghong Wu, Ashna Zaheer, Joel Haas, Kiwhoon Lee, Baoli Yang
Diminished Cytokine And Chemokine Expression In The Central Nervous System Of Gmf-Deficient Mice With Experimental Autoimmune Encephalomyelitis, Asgar Zaheer, Shailendra Sahu, Yanghong Wu, Ashna Zaheer, Joel Haas, Kiwhoon Lee, Baoli Yang
Baoli Yang
No abstract provided.
Loss Of Vac14, A Regulator Of The Signaling Lipid Phosphatidylinositol 3,5-Bisphosphate, Results In Neurodegeneration In Mice, Yanling Zhang, Sergey Zolov, Clement Chow, Shalom Slutsky, Simon Richardson, Robert Piper, Baoli Yang, Johnathan Nau, Randal Westric, Sean Morrison, Miriam Meisler, Lois Weisman
Loss Of Vac14, A Regulator Of The Signaling Lipid Phosphatidylinositol 3,5-Bisphosphate, Results In Neurodegeneration In Mice, Yanling Zhang, Sergey Zolov, Clement Chow, Shalom Slutsky, Simon Richardson, Robert Piper, Baoli Yang, Johnathan Nau, Randal Westric, Sean Morrison, Miriam Meisler, Lois Weisman
Baoli Yang
No abstract provided.
Renal Tubular Nedd4-2 Deficiency Causes Ncc-Mediated Salt-Dependent Hypertension, Caroline Ronzaud, Dominique Loffing-Cueni, Pierrette Hausel, Anne Debonneville, Sumedha Malsure, Nicole Fowler-Jaeger, Natasha Boase, Romain Perrier, Marc Maillard, Baoli Yang, John Stokes, Robert Koesters, Sharad Kumar, Edith Hummler, Johannes Loffing, Olivier Staub
Renal Tubular Nedd4-2 Deficiency Causes Ncc-Mediated Salt-Dependent Hypertension, Caroline Ronzaud, Dominique Loffing-Cueni, Pierrette Hausel, Anne Debonneville, Sumedha Malsure, Nicole Fowler-Jaeger, Natasha Boase, Romain Perrier, Marc Maillard, Baoli Yang, John Stokes, Robert Koesters, Sharad Kumar, Edith Hummler, Johannes Loffing, Olivier Staub
Baoli Yang
No abstract provided.
Mice Defective In Trpm6 Show Embryonic Mortality And Neural Tube Defects, Roxanne Walder, Baoli Yang, John Stokes, Patricia Kirby, Xiao Cao, Peijun Shi, Charles Searby, Russell Husted, Val Sheffield
Mice Defective In Trpm6 Show Embryonic Mortality And Neural Tube Defects, Roxanne Walder, Baoli Yang, John Stokes, Patricia Kirby, Xiao Cao, Peijun Shi, Charles Searby, Russell Husted, Val Sheffield
Baoli Yang
No abstract provided.
B Cell-Specific Traf3-Deficient Mice Exhibit Expanded B Cell Compartments In Secondary Lymphoid Organs, Ping Xie, Laura Stunz, Karen Larison, Baoli Yang, Gail Bishop
B Cell-Specific Traf3-Deficient Mice Exhibit Expanded B Cell Compartments In Secondary Lymphoid Organs, Ping Xie, Laura Stunz, Karen Larison, Baoli Yang, Gail Bishop
Baoli Yang
No abstract provided.
Glia Maturation Factor Overexpression In Neuroblastoma Cells Activates Glycogen Synthase Kinase-3 Beta And Caspase-3, Asgar Zaheer, Scott Knight, Ashna Zaheer, Marcus Ahrens, Shailendra Sahu, Baoli Yang
Glia Maturation Factor Overexpression In Neuroblastoma Cells Activates Glycogen Synthase Kinase-3 Beta And Caspase-3, Asgar Zaheer, Scott Knight, Ashna Zaheer, Marcus Ahrens, Shailendra Sahu, Baoli Yang
Baoli Yang
No abstract provided.
A Common Disease-Associated Missense Mutation In Alpha-Sarcoglycan Fails To Cause Muscular Dystrophy In Mice, Kazuhiro Kobuke, Federica Piccolo, Keith Garringer, Steven Moore, Eileen Sweezer, Baoli Yang, Kevin Campbell
A Common Disease-Associated Missense Mutation In Alpha-Sarcoglycan Fails To Cause Muscular Dystrophy In Mice, Kazuhiro Kobuke, Federica Piccolo, Keith Garringer, Steven Moore, Eileen Sweezer, Baoli Yang, Kevin Campbell
Baoli Yang
No abstract provided.
Impaired Motor Performance And Learning In Glia Maturation Factor-Knockout Mice, R. Lim, A. Zaheer, H. Khosravi, J. Freeman, H. Halverson, J. Wemmie, Baoli Yang
Impaired Motor Performance And Learning In Glia Maturation Factor-Knockout Mice, R. Lim, A. Zaheer, H. Khosravi, J. Freeman, H. Halverson, J. Wemmie, Baoli Yang
Baoli Yang
No abstract provided.
The Role Of Ubiquitination In Hepcidin-Independent And Hepcidin-Dependent Degradation Of Ferroportin (Retraction Of Vol 14, Pg 635, 2011), Ivana De Domenico, Eric Lo, Baoli Yang, Tamara Korolnek, Iqbal Hamza, Diane Ward, Jerry Kaplan
The Role Of Ubiquitination In Hepcidin-Independent And Hepcidin-Dependent Degradation Of Ferroportin (Retraction Of Vol 14, Pg 635, 2011), Ivana De Domenico, Eric Lo, Baoli Yang, Tamara Korolnek, Iqbal Hamza, Diane Ward, Jerry Kaplan
Baoli Yang
No abstract provided.
Nedd4-2 Modulates Renal Na+-Cl- Cotransporter Via The Aldosterone-Sgk1-Nedd4-2 Pathway, Juan Pablo Arroyo, Dagmara Lagnaz, Caroline Ronzaud, Norma Vazquez, Benjamin Ko, Lauren Moddes, Dorothee Ruffieux-Daidie, Pierrette Hausel, Robert Koesters, Baoli Yang, John Stokes, Robert Hoover, Gerardo Gamba, Olivier Staub
Nedd4-2 Modulates Renal Na+-Cl- Cotransporter Via The Aldosterone-Sgk1-Nedd4-2 Pathway, Juan Pablo Arroyo, Dagmara Lagnaz, Caroline Ronzaud, Norma Vazquez, Benjamin Ko, Lauren Moddes, Dorothee Ruffieux-Daidie, Pierrette Hausel, Robert Koesters, Baoli Yang, John Stokes, Robert Hoover, Gerardo Gamba, Olivier Staub
Baoli Yang
No abstract provided.
Tumor Necrosis Factor Receptor-Associated Factor 3 Is A Critical Regulator Of B Cell Homeostasis In Secondary Lymphoid Organs, Ping Xie, Laura Stunz, Karen Larison, Baoli Yang, Gail Bishop
Tumor Necrosis Factor Receptor-Associated Factor 3 Is A Critical Regulator Of B Cell Homeostasis In Secondary Lymphoid Organs, Ping Xie, Laura Stunz, Karen Larison, Baoli Yang, Gail Bishop
Baoli Yang
No abstract provided.
Loss Of Mxin Alpha, An Intercalated Disk Protein, Results In Cardiac Hypertrophy And Cardiomyopathy With Conduction Defects, Elisabeth Gustafson-Wagner, Haley Sinn, Yen-Lin Chen, Da-Zhi Wang, Rebecca Reiter, Jenny Lin, Baoli Yang, Roger Williamson, Ju Chen, Cheng-I Lin, Jim J. -C Lin
Loss Of Mxin Alpha, An Intercalated Disk Protein, Results In Cardiac Hypertrophy And Cardiomyopathy With Conduction Defects, Elisabeth Gustafson-Wagner, Haley Sinn, Yen-Lin Chen, Da-Zhi Wang, Rebecca Reiter, Jenny Lin, Baoli Yang, Roger Williamson, Ju Chen, Cheng-I Lin, Jim J. -C Lin
Baoli Yang
No abstract provided.
Identification And Functional Analysis Of The Vision-Specific Bbs3 (Arl6) Long Isoform, Pamela Pretorius, Lisa Baye, Darryl Nishimura, Charles Searby, Kevin Bugge, Baoli Yang, Robert Mullins, Edwin Stone, Val Sheffield, Diane Slusarski
Identification And Functional Analysis Of The Vision-Specific Bbs3 (Arl6) Long Isoform, Pamela Pretorius, Lisa Baye, Darryl Nishimura, Charles Searby, Kevin Bugge, Baoli Yang, Robert Mullins, Edwin Stone, Val Sheffield, Diane Slusarski
Baoli Yang
No abstract provided.
Physiological Significance Of Two Common Haplotypes Of Human Angiotensinogen Using Gene Targeting In The Mouse, B. Cvetkovic, H. Keen, X. Zhang, D. Davis, Baoli Yang, C. Sigmund
Physiological Significance Of Two Common Haplotypes Of Human Angiotensinogen Using Gene Targeting In The Mouse, B. Cvetkovic, H. Keen, X. Zhang, D. Davis, Baoli Yang, C. Sigmund
Baoli Yang
No abstract provided.