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Articles 31 - 36 of 36
Full-Text Articles in Environmental Health
Risk And Consequence Analysis Focused On Biota Transfers Potentially Associated With Surface Water Diversions Between The Missouri River And Red River Basins, Greg Linder, Ed Little, Lynne Johnson, Chad Vishy, Bruce Peacock, Heather Goeddecke
Risk And Consequence Analysis Focused On Biota Transfers Potentially Associated With Surface Water Diversions Between The Missouri River And Red River Basins, Greg Linder, Ed Little, Lynne Johnson, Chad Vishy, Bruce Peacock, Heather Goeddecke
United States Geological Survey: Publications
Section 1 provides a brief overview of the project, including a cursory summary of the history of the “Garrison Diversion” and how that history relates to this work focused on the analysis of risks and consequences potentially associated with interbasin biota transfers. The present study was initiated under the auspices of the Dakota Water Resources Act (DWRA) of 2000, which directed the Secretary of the Interior to conduct a comprehensive study of the water quality and quantity needs of the Red River Valley and the options for meeting those needs. As such, the Bureau of Reclamation (Reclamation) requested technical support …
Atrazine Runoff In The Blue River Basin: Geomorphology, Rainfall, And Agronomic Practices, Kundan Dhakal
Atrazine Runoff In The Blue River Basin: Geomorphology, Rainfall, And Agronomic Practices, Kundan Dhakal
Department of Agronomy and Horticulture: Dissertations, Theses, and Student Research
Atrazine concentrations in the Big Blue River Basin (BRB) in Nebraska and Kansas periodically exceed the U.S. EPA Maximum Contaminant Level (MCL) of 3 µg L-1. The present study is focused on watershed variables influencing surface runoff of atrazine. The assessment includes the impact of corn and sorghum planting progress (indicating atrazine application), rainfall, antecedent soil water content, and soil restrictive layer on stream-measured weekly atrazine load in independent BRB subwatersheds for 1997 - 2004. Maximum atrazine loading occurred after most of the corn had been planted but during sorghum planting from mid-May to early June, immediately following …
Escherichia Coli O157:H7 In Free-Ranging Deer In Nebraska, David G. Renter, Jan M. Sargeant, Scott E. Hygnstrom, Jeff D. Hoffmann, Jerry R. Gillespie
Escherichia Coli O157:H7 In Free-Ranging Deer In Nebraska, David G. Renter, Jan M. Sargeant, Scott E. Hygnstrom, Jeff D. Hoffmann, Jerry R. Gillespie
School of Natural Resources: Faculty Publications
In order to determine the prevalence and distribution of the human pathogen, Escherichia coli O157:H7, in free-ranging deer, hunters were asked to collect and submit fecal samples from deer harvested during a regular firearm season (14–22 November 1998). Prior to the season, 47% of the hunters with permits in the southeastern Nebraska (USA) study area indicated a willingness to participate in the study. Approximately 25% of successful hunters in the area submitted deer fecal samples. Escherichia coli O157:H7 was cultured from four (0.25%) of 1,608 total samples submitted. All of the fecal samples that were properly identified (1,426) and all …
Can We Guarantee The Safety Of Genetically Engineered Organisms In The Environment?, Kathleen H. Keeler, Frances E. Sharples
Can We Guarantee The Safety Of Genetically Engineered Organisms In The Environment?, Kathleen H. Keeler, Frances E. Sharples
School of Biological Sciences: Faculty Publications
Can we guarantee the safety of genetically engineered organisms in the environment? To anticipate my remarks, the answer to the title question is “No, we cannot guarantee the safety of genetically engineered organisms released into the environment.” Indeed, it is a tenet of the scientific method that nothing can be proved, only disproved. Thus, we can never show that a release will be safe. We could only show that it would be unsafe, if that were the case. However, if the question is posed differently, for example, can we safely release genetically engineered organisms into the environment?, the answer is …
Lead Poisoning Of Sandhill Cranes (Grus Canadensis), R. M. Windingstad, S. M. Kerr, L. N. Locke, J. J. Hurt
Lead Poisoning Of Sandhill Cranes (Grus Canadensis), R. M. Windingstad, S. M. Kerr, L. N. Locke, J. J. Hurt
Papers in Ornithology
Two wild and two captive sandhill cranes (Grus canadensis) were diagnosed by National Wildlife Health Laboratory personnel as having died from lead toxicity. Ingestion of lead fishing weights by the wild cranes and of unspent .22 caliber shell cartidges by the captive cranes were responsible for these deaths. One crane force-fed lead pellets showed an increase of blood lead levels from 0.77 ppm to 23.8 ppm (wet weight) just before its death 15 days following exposure. Liver lead concentrations of sandhill cranes dying of causes other than lead toxicity are presented.
The Pharmacology Of Rodenticides, S. A. Peoples
The Pharmacology Of Rodenticides, S. A. Peoples
Vertebrate Pest Conference Proceedings: 4th (1970)
The compounds used as rodenticides are tremendously varied in their chemical structure and mechanism of action. With a few exceptions, these agents are generally poisonous to all animals, including man, and a great deal of study has been directed to their toxicity in animals other than rodents. However, the development of new compounds as Norbormide and certain antifertility drugs which are highly selective in their action may justify the hope that the ideal rodenticide free of secondary toxic hazards will soon be available. Until this happy announcement is made, a review of the pharmacology of the older compounds is in …