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Full-Text Articles in Environmental Public Health
Low-Cost Pm2.5 Sensors Can Help Identify Driving Factors Of Poor Air Quality And Benefit Communities, Tim Keyes, Rea Domingo, Samantha Dynowski, Royal Graves, Martha Klein, Melissa Leonard, John Pilgrim, Alison Sanchirico, Kate Trinkaus
Low-Cost Pm2.5 Sensors Can Help Identify Driving Factors Of Poor Air Quality And Benefit Communities, Tim Keyes, Rea Domingo, Samantha Dynowski, Royal Graves, Martha Klein, Melissa Leonard, John Pilgrim, Alison Sanchirico, Kate Trinkaus
WCBT Faculty Publications
Air quality is critical for public health. Residents rely chiefly on government agencies such as the Environmental Protection Agency (EPA) in the United States to establish standards for the measurement of harmful contaminants including ozone, sulfur dioxide, carbon monoxide, volatile organic chemicals (VOCs), and fine particulate matter at or below 2.5 μm. According to the California Air Resources Board [1], “short-term PM2.5 exposure (up to 24-h duration) has been associated with premature mortality, increased hospital admissions for heart or lung causes, acute and chronic bronchitis, asthma attacks, emergency room visits, respiratory symptoms, and restricted activity days”. While public agency resources …
Association Between Residential Exposure To Air Pollution And Incident Coronary Heart Disease Is Not Mediated By Leukocyte Telomere Length: A Uk Biobank Study, Chia-Ling Kuo, Rui Liu, Lucas Da Cunha Godoy, Luke C. Pilling, Richard H. Fortinsky, Doug Brugge
Association Between Residential Exposure To Air Pollution And Incident Coronary Heart Disease Is Not Mediated By Leukocyte Telomere Length: A Uk Biobank Study, Chia-Ling Kuo, Rui Liu, Lucas Da Cunha Godoy, Luke C. Pilling, Richard H. Fortinsky, Doug Brugge
Health Science Faculty Publications
Higher air pollution exposure and shorter leukocyte telomere length (LTL) are both associated with increased risk of coronary heart disease (CHD), and share plausible mechanisms, including inflammation. LTL may serve as a biomarker of air pollution exposure and may be intervened with to reduce the risk of CHD. To the best of our knowledge, we are the first to test the mediation effect of LTL in the relationship between air pollution exposure and incident CHD. Using the UK Biobank (UKB) data (n = 317,601), we conducted a prospective study linking residential air pollution exposure (PM2.5, PM10, NO2, NOx) and …