Open Access. Powered by Scholars. Published by Universities.®
- Discipline
- Keyword
-
- Animals (4)
- Mice (3)
- Microglia (3)
- Motoneuron (3)
- Mouse (3)
-
- Attention (2)
- CRAC channel (2)
- Ca2+ channel (2)
- Carotid body (2)
- Caspases (2)
- Cation channel (2)
- FMRI (2)
- Lymphocyte (2)
- Metabolism (2)
- Mitochondria (2)
- Nerve (2)
- Neurofeedback (2)
- Neuromodulation (2)
- Neuromuscular Junction (2)
- Neurons (2)
- Plasticity (2)
- Primary auditory cortex (2)
- ALS (1)
- Accessory Proteins (1)
- Acetylcholine Receptor (1)
- Acidification (1)
- Action potential (1)
- Activation (1)
- Adults (1)
- Aging (1)
Articles 91 - 92 of 92
Full-Text Articles in Neurosciences
Electrophysiology Of Flounder Intestinal Mucosa. Ii. Relation Of The Electrical Potential Profile To Coupled Nacl Absorption, Dan R. Halm, Edward J. Krasny Jr., Raymond A. Frizzell
Electrophysiology Of Flounder Intestinal Mucosa. Ii. Relation Of The Electrical Potential Profile To Coupled Nacl Absorption, Dan R. Halm, Edward J. Krasny Jr., Raymond A. Frizzell
Neuroscience, Cell Biology & Physiology Faculty Publications
We characterized the hyperpolarization of the electrical potential profile of flounder intestinal cells that accompanies inhibition of NaCl cotransport. Several observations indicate that hyperpolarization of ψa and ψb (Δψa,b) results from inhibition of NaCl entry across the apical membrane: (a) the response was elicited by replacement of mucosal solution Cl or Na by nontransported ions, and (b) mucosal bumetanide or serosal cGMP, inhibitors of NaCl influx, elicited Δψa,b and decreased the transepithelial potential (ψt) in parallel. Regardless of initial values, ψa and ψb approached the equilibrium potential for K (EK …
Metabolism In Preimplantation Mouse Embryos, Harry M. Weitlauf, Gary L. Nieder
Metabolism In Preimplantation Mouse Embryos, Harry M. Weitlauf, Gary L. Nieder
Neuroscience, Cell Biology & Physiology Faculty Publications
The ability of preimplantation mouse embryos to utilize glucose oxidatively is controlled, in part at least, at the level of glycolysis. Various experimental observations are reviewed that indicate the regulatory mechanism in delayed implanting blastocysts involves the classic negative allosteric feedback of high levels of ATP on phosphofructokinase while the situation in 2-cell embryos appears to be more complicated. That is, in addition to the usual negative effect of ATP and citrate on phosphofructokinase, there appears to be a modification of hexokinase that prevents phosphorylation of adequate amounts of glucose and results in low levels of fructose-6-phosphate at the 2-cell …