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Effects Of Mortality Risk And Growth On A Model Of Reproductive Effort: Why The Shine And Schwarzkopf Model Is Not General, Peter Niewiarowski, Arthur Dunham
Effects Of Mortality Risk And Growth On A Model Of Reproductive Effort: Why The Shine And Schwarzkopf Model Is Not General, Peter Niewiarowski, Arthur Dunham
Dr. Peter H. Niewiarowski
Using data and reanalysis of a model published by Shine and Schwarzkopf (1992), we reject the two unsubstantiated assertions made by Shine et al. (1996) about modeling the evolution of reproductive effort in squamate reptiles.
The Evolution Of Copulation Frequency And The Mechanisms Of Reproduction In Male Anolis Lizards, Michele Johnson, Maria Lopez, Tara Whittle, Bonnie Kircher, A Dill, Divina Varghese, Juli Wade
The Evolution Of Copulation Frequency And The Mechanisms Of Reproduction In Male Anolis Lizards, Michele Johnson, Maria Lopez, Tara Whittle, Bonnie Kircher, A Dill, Divina Varghese, Juli Wade
Michele A Johnson
The evolution of many morphological structures is associated with the behavioral context of their use, particularly for structures involved in copulation. Yet, few studies have considered evolutionary relationships among the integrated suite of structures associated with male reproduction. In this study, we examined nine species of lizards in the genus Anolis to determine whether larger copulatory morphologies and higher potential for copulatory muscle performance evolved in association with higher copulation rates. In 10–12 adult males of each species, we measured the size of the hemipenes and related muscles, the seminiferous tubules in the testes, and the renal sex segments in …
P19ink4d And P18ink4c Cyclin-Dependent Kinase Inhibitors In The Male Reproductive Axis, Gregory Buchold, Patricia Magyar, Ramamani Arumugam, Mary Lee, Deborah O'Brien
P19ink4d And P18ink4c Cyclin-Dependent Kinase Inhibitors In The Male Reproductive Axis, Gregory Buchold, Patricia Magyar, Ramamani Arumugam, Mary Lee, Deborah O'Brien
Mary M. Lee
The loss of the cyclin-dependent kinase inhibitors (CKIs) p18(Ink4c) and p19(Ink4d) leads to male reproductive defects (Franklin et al., 1998. Genes Dev 12: 2899-2911; Zindy et al., 2000. Mol Cell Biol 20: 372-378; Zindy et al., 2001. Mol Cell Biol 21: 3244-3255). In order to assess whether these inhibitors directly or indirectly affect male germ cell differentiation, we examined the expression of p18(Ink4c) and p19(Ink4d) in spermatogenic and supporting cells in the testis and in pituitary gonadotropes. Both p18(Ink4c) and p19(Ink4d) are most abundant in the testis after 18 days of age and are expressed in purified populations of spermatogenic …