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Full-Text Articles in Life Sciences
Preliminary Observations On The Usefulness Of Hinge Structures For Identification Of Bivalve Larvae, R. Lutz, J. Goodsell, M. Castagna, S. Chapman, C. Newell, H. Hidu, R. Mann, Et Al
Preliminary Observations On The Usefulness Of Hinge Structures For Identification Of Bivalve Larvae, R. Lutz, J. Goodsell, M. Castagna, S. Chapman, C. Newell, H. Hidu, R. Mann, Et Al
VIMS Articles
Difficulties associated with discrimination of bivalve larvae isolated from plankton samples have long hampered both applied and basic research efforts in estuarine and open coastal marine environments. The vast majority of practical barriers to identification of larval bivalves may be eliminated through routine optical microscopic examination of the hinge apparatus of disarticulated larval shells. Representative micrographs of various ontogenetic stages of larval hinge development are presented for 12 genera (Mytilus, Geukensia, Crassostrea, Placopecten. Argopecten. Mya, Spisula, Mulinia, Ensis, Arca, Arctica. and Mercenaria) from 9 bivalve superfamilies (Mytilacea, Ostreacea. Pectinacea, Myacea, Mactracea. Solenacea, Arcacea, Arcticacea. and Veneracea). The larval hinge apparatus …
Anaerobic Mortalities Of Oysters In Virginia Caused By Low Salinities, J. D. Andrews
Anaerobic Mortalities Of Oysters In Virginia Caused By Low Salinities, J. D. Andrews
VIMS Articles
Oysters on natural beds in the upper seed area of the James River died anaerobically in the winter and early spring of 1979-80 during prolonged exposure to fresh water and low salinities (< 5 ppt). Heavy rains in .the fall of 1979 combined with the usual winter-spring runoff to produce low salinities. Oysters in trays were transplanted in late March and early April to six high-salinity areas where mortalities were found a month later. The oysters died slowly within closed shells because they were unable to feed and respire in the nearly fresh water. This produced a strong, malodorus stench and blackened shell margins that are characteristic of anaerobiotic decay. Similar phenomena occurred previously in the Rappahannock River about 1 May during several wet years during the past three decades. At depths of 5 to 6 m, dissolved oxygen was depleted and everything on the bottom became black with iron and other heavy metal sulfides. Dead oysters were not discovered until June after waters had become aerobic again.
Epizootiology Of Late Summer And Fall Infections Of Oysters By Haplosporidium Nelsoni, And Comparisons To Annual Life Cycle Of Haplosporidium Costalis, A Typical Haplosporidan, J. D. Andrews
VIMS Articles
The two haplosporidan parasites that cause diseases of oysters along the middle North Atlantic coast of North America differ in their habitats, in timing of oyster mortalities, and in their adaptations to the host. Haplosporidium nelsoni (MSX) kills oysters throughout the year over a wide range of salinities (about 15 to 30 ppt). It has a long infective period of nearly 6 months. This pathogen rarely completes sporulation in its life cycle in oysters. It is highly pathogenic and exhibits irregular activity suggesting that it is poorly adapted to the host species. In contrast, Haplosporidium costalis (SSO) has a short, …
Larval Development Of Citharichthys-Cornutus, Citharichthys-Gymnorhinus, Citharichthys-Spilopterus, And Etropus-Crossotus (Bothidae), With Notes On Larval Occurrence, John W. Tucker Jr.
Larval Development Of Citharichthys-Cornutus, Citharichthys-Gymnorhinus, Citharichthys-Spilopterus, And Etropus-Crossotus (Bothidae), With Notes On Larval Occurrence, John W. Tucker Jr.
VIMS Articles
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