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Series

COVID-19

University of Texas at El Paso

Articles 1 - 4 of 4

Full-Text Articles in Nursing

Herbal Medicines In The Treatment Of Coronavirus Disease 2019 (Covid-19), Audrey C. Tolouian, Maryam Khosravian, Hedieh Ragati Haghi, Alireza Bolourian, Zahra Mojtahedi, Masoumeh Asgharpour, Amirhesam Alirezaei Nov 2020

Herbal Medicines In The Treatment Of Coronavirus Disease 2019 (Covid-19), Audrey C. Tolouian, Maryam Khosravian, Hedieh Ragati Haghi, Alireza Bolourian, Zahra Mojtahedi, Masoumeh Asgharpour, Amirhesam Alirezaei

Nursing Faculty Publications

Coronavirus disease 2019 (COVID-19), caused by a novel coronavirus, started in livestock within the markets of Wuhan, China and was consequently spread around the world. The virus has been rapidly spread worldwide due to the outbreak. COVID-19 is the third serious coronavirus outbreak in less than 20 years after Severe Acute Respiratory Syndrome (SARS) in 2003 and Middle East Respiratory Syndrome (MERS) in 2012. The novel virus has a nucleotide identity closer to that of the SARS coronavirus than that of the MERS coronavirus. Since there is still no vaccine, the main ways to improve personal immunity against this disease …


The Pandemic Need For Mindfulness, Audrey C. Tolouian, Melissa Wholeben Aug 2020

The Pandemic Need For Mindfulness, Audrey C. Tolouian, Melissa Wholeben

Nursing Faculty Publications

In a trauma facility, it is important that there be training for the healthcare providers that gives them tools to manage their internal reactions to the day-to-day events, as well as create a safe space and encourage a sense of community. For healthcare workers in high-trauma areas- such as the new COVID-19 units, post-traumatic stress syndrome (PTSD) has been on the rise, in 2011, 14% of nurses were diagnosed, in 2019 an estimated 40% of nurses suffer from the disorder. With the small amount of 18% of nurses feeling they are prepared with proficient self-care skills to manage the effects …


The Footprint Of Androgen Sensitive Serine Protease (Tmprss2) In Gender Mortality With Covid-19, Sepideh Zununi Vahed, Shahram Ghiyasvand, Ramin Tolouian, Hamid Noshad, Audrey C. Tolouian, Mohammadali Mohajel Shoja, Mohammadreza Ardalan May 2020

The Footprint Of Androgen Sensitive Serine Protease (Tmprss2) In Gender Mortality With Covid-19, Sepideh Zununi Vahed, Shahram Ghiyasvand, Ramin Tolouian, Hamid Noshad, Audrey C. Tolouian, Mohammadali Mohajel Shoja, Mohammadreza Ardalan

Nursing Faculty Publications

Male gender is an obvious risk factor for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection and mortality rate is higher in men than women. Undoubtedly, gender-related behavioral factors, such as higher amounts of smoking, alcohol consumption, and biological differences in immune systems could make males more vulnerable. The role of androgen-responsive elements (AREs) of transmembrane serine proteases type II (TMPRSS2) gene as one of the major players of male dominancy in severe COVID-19 infection has been under appreciated and needs to be clarified.


Covid-19 Interactions With Angiotensin-Converting Enzyme 2 (Ace2) And The Kinin System; Looking At A Potential Treatment, Ramin Tolouian, Sepideh Zununi Vahed, Shahram Ghiyasvand, Audrey C. Tolouian, Mohammadreza Ardalan Mar 2020

Covid-19 Interactions With Angiotensin-Converting Enzyme 2 (Ace2) And The Kinin System; Looking At A Potential Treatment, Ramin Tolouian, Sepideh Zununi Vahed, Shahram Ghiyasvand, Audrey C. Tolouian, Mohammadreza Ardalan

Nursing Faculty Publications

The novel coronavirus disease 2019 (COVID-19) is a rapidly expanding infection around the world. The world Health Organization (WHO) in March 2020 announced the Coronavirus pandemic. This infection causes many deaths on daily basis. Therapeutic options are currently limited. It is revealed that COVID-19 binds to human angiotensin-converting enzyme 2 (ACE2) to enter the host cells. One of the activities of ACE2 is hydrolyzing the active bradykinin metabolite [des-Arg973] BK (DABK). A decreased activity or reducing expression of ACE2 by the virus impairs the inactivation of DABK. This enhances its signaling through the bradykinin B1 receptor (BKB1R) and could lead …