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Long-Term Effects Of 3,4- Methylenedioxymethamphetamine (Mdma) On Serotonergic And Dopaminergic Functioning, Jodi Lynn Kohutek
Long-Term Effects Of 3,4- Methylenedioxymethamphetamine (Mdma) On Serotonergic And Dopaminergic Functioning, Jodi Lynn Kohutek
Theses Digitization Project
Methylenedioxymethamphetamine (MDMA) popularly known as "Ecstasy" continues to gain popularity as a recreational drug that has been shown to increase serotonin and dopamine levels. The present study has demonstrated that repeated exposure to MDMA produces long-term damage to serotonergic and dopaminergic neurons in various regions of the rat brain.
Induction And Expression Of Cocaine-Induced Behavioral Sensitization: Modulation By A Partial D₂-Like Agonist, Janet Marie Sibole
Induction And Expression Of Cocaine-Induced Behavioral Sensitization: Modulation By A Partial D₂-Like Agonist, Janet Marie Sibole
Theses Digitization Project
The purpose of this study was to investigate whether a partial D₂-like dopamine agonist (i.e. terguride) would block the induction or expression of cocaine-induced behavioral sensitization in pre-weanling rats. The ability of terguride to induce behavioral sensitization was also examined, as partial D₂-like agonists have agonistic actions in cases of low dopaminergic tone.
Ultrasonic Vocalizations Of Preweanling Rats The Interaction Of K-Opioid And A₂-Noradrenergic Systems, Arbi Nazarian
Ultrasonic Vocalizations Of Preweanling Rats The Interaction Of K-Opioid And A₂-Noradrenergic Systems, Arbi Nazarian
Theses Digitization Project
No abstract provided.
Role Of The Dopamine D₁-Like Receptor In Amphetamine-Induced Behavioral Sensitization: A Study Using Dopamine D₁A-Receptor Deficient Mice, Patrick Eugene Karper
Role Of The Dopamine D₁-Like Receptor In Amphetamine-Induced Behavioral Sensitization: A Study Using Dopamine D₁A-Receptor Deficient Mice, Patrick Eugene Karper
Theses Digitization Project
The ability of the indirect dopamine agonist, amphetamine, to produce behavioral sensitization was assessed in adult D₁A-deficient and wild-type mice. It was originally predicted that : 1) dopamine (DA) D₁-like receptors are necessary for the occurrence of short- and long-term amphetamine-induced behavioral sensitization, 2) DA D₁-like receptors are necessary for environmental conditioning factors associated with amphetamine-induced behavioral sensitiazation, and 3) DA D₅ receptors are required for amphetamine-induced behavioral sensitization. Locomotor activity and sterotyped sniffing were assessed in each of three experiments.
The Effects Of Kappa Opioid And Dopamine Agonists On Unconditioned Behaviors And Fos Immunoreactivity In Preweanling And Adult Rats, Marcus Alan Duke
The Effects Of Kappa Opioid And Dopamine Agonists On Unconditioned Behaviors And Fos Immunoreactivity In Preweanling And Adult Rats, Marcus Alan Duke
Theses Digitization Project
No abstract provided.
The Effects Of Dopamine D1 And D2 Antagonists On Cocaine-Induced Cpp In Preweanling Rats, Douglas L. Pruitt
The Effects Of Dopamine D1 And D2 Antagonists On Cocaine-Induced Cpp In Preweanling Rats, Douglas L. Pruitt
Theses Digitization Project
The effects of dopamine D1 and D2 antagonists on conditioned place preference (CPP) and locomotor activity were assessed. A total of three experiments were conducted. In each experiment there were two conditioning days followed by a test day. The results indicate that DA D1 and D2 receptors have distictly different roles in the mediation of behavior.
The Effect Of Phenol Denervation Of The Hepatic Portal Vein Nerves On Taste Aversion Learning, Deborah Jane Hooks
The Effect Of Phenol Denervation Of The Hepatic Portal Vein Nerves On Taste Aversion Learning, Deborah Jane Hooks
Theses Digitization Project
No abstract provided.
Effects Of Dopamine D1 And D2 Receptor Inactivation On Locomotor Activity And Sniffing In 11- And 17-Day-Old Rats, Monja Mestlin
Effects Of Dopamine D1 And D2 Receptor Inactivation On Locomotor Activity And Sniffing In 11- And 17-Day-Old Rats, Monja Mestlin
Theses Digitization Project
No abstract provided.