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Transgene And Transposon Silencing In Chlamydomonas Reinhardtii By A Deah-Box Rna Helicase, Dancia Wu-Scharf, Byeong-Ryool Jeong, Chaomei Zhang, Heriberto D. Cerutti
Transgene And Transposon Silencing In Chlamydomonas Reinhardtii By A Deah-Box Rna Helicase, Dancia Wu-Scharf, Byeong-Ryool Jeong, Chaomei Zhang, Heriberto D. Cerutti
Center for Plant Science Innovation: Faculty and Staff Publications
The molecular mechanism(s) responsible for posttranscriptional gene silencing and RNA interference remain poorly understood. We have cloned a gene (Mut6) from the unicellular green alga Chlamydomonas reinhardtii that is required for the silencing of a transgene and two transposon families. Mut6 encodes a protein that is highly homologous to RNA helicases of the DEAH-box family. This protein is necessary for the degradation of certain aberrant RNAs, such as improperly processed transcripts, which are often produced by transposons and some transgenes.
Pseudomonas Syringae Hrp Type Iii Secretion System And Effector Proteins, Alan Collmer, Jorge L. Badel, Amy O. Charkowski, Wen-Ling Deng, Derrick E. Fouts, Adela R. Ramos, Amos H. Rehm, Deborah M. Anderson, Olaf Schneewind, Karin V. Van Dijk, James R. Alfano
Pseudomonas Syringae Hrp Type Iii Secretion System And Effector Proteins, Alan Collmer, Jorge L. Badel, Amy O. Charkowski, Wen-Ling Deng, Derrick E. Fouts, Adela R. Ramos, Amos H. Rehm, Deborah M. Anderson, Olaf Schneewind, Karin V. Van Dijk, James R. Alfano
Center for Plant Science Innovation: Faculty and Staff Publications
Pseudomonas syringae is a member of an important group of Gram-negative bacterial pathogens of plants and animals that depend on a type III secretion system to inject virulence effector proteins into host cells. In P. syringae, hrpyhrc genes encode the Hrp (type III secretion) system, and avirulence (avr) and Hrpdependent outer protein (hop) genes encode effector proteins. The hrpyhrc genes of P. syringae pv syringae 61, P. syringae pv syringae B728a, and P. syringae pv tomato DC3000 are flanked by an exchangeable effector locus and …
The Pseudomonas Syringae Hrp Pathogenicity Island Has A Tripartite Mosaic Structure Composed Of A Cluster Of Type Iii Secretion Genes Bounded By Exchangeable Effector And Conserved Effector Loci That Contribute To Parasitic Fitness And Pathogenicity In Plants, James R. Alfano, Amy O. Charkowski, Wen-Ling Deng, Jorge L. Badel, Tanja Petnicki- Ocwieja, Karin Van Dijk, Alan Collmer
The Pseudomonas Syringae Hrp Pathogenicity Island Has A Tripartite Mosaic Structure Composed Of A Cluster Of Type Iii Secretion Genes Bounded By Exchangeable Effector And Conserved Effector Loci That Contribute To Parasitic Fitness And Pathogenicity In Plants, James R. Alfano, Amy O. Charkowski, Wen-Ling Deng, Jorge L. Badel, Tanja Petnicki- Ocwieja, Karin Van Dijk, Alan Collmer
Center for Plant Science Innovation: Faculty and Staff Publications
The plant pathogenic bacterium Pseudomonas syringae is divided into pathovars differing in host specificity, with P. syringae pv. syringae (Psy) and P. syringae pv. tomato (Pto) representing particularly divergent pathovars. P. syringae hrp/hrc genes encode a type III protein secretion system that appears to translocate Avr and Hop effector proteins into plant cells. DNA sequence analysis of the hrp/hrc regions in Psy 61, Psy B728a, and Pto DC3000 has revealed a Hrp pathogenicity island (Pai) with a tripartite mosaic structure. The hrp/hrc gene cluster is conserved in all three strains and is flanked by a unique exchangeable …